This is an agent-based model with two types of agents: customers and insurers. Insurers are price-takers who choose how much to spend on their service quality, and customers evaluate insurers based on premium, brand preference, and their perceived service quality. Customers are also connected in a small-world network and may share their opinions with their network.

The ABM contains two types of agents: insurers and customers. These act within the environment of a motor insurance market. At each simulation, the model undergoes the following steps:

1. Network generation: At the start of the simulation, the model generates a small world network of social links between the customers, and randomly assigns each customer to an initial insurer
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In this model, the spread of a virus disease in a network consisting of school pupils, employed, and umemployed people is simulated. The special feature in this model is the distinction between different types of links: family-, friends-, school-, or work-links. In this way, different governmental measures can be implemented in order to decelerate or stop the transmission.

The Agent-Based Wildfire Simulation Environment (ABWiSE) translates the concept of a moving fire front as a set of mobile fire agents that respond to, and interact with, vegetation, wind, and terrain. Presently, the purpose of ABWiSE is to explore how ABM, using simple interactions between agents and a simple atmospheric feedback model, can simulate emergent fire spread patterns.

At the heart of a study of Social-Ecological Systems, this model is built by coupling together two independently developed models of social and ecological phenomena. The social component of the model is an abstract model of interactions of a governing agent and several user agents, where the governing agent aims to promote a particular behavior among the user agents. The ecological model is a spatial model of spread of the Mountain Pine Beetle in the forests of British Columbia, Canada. The coupled model allowed us to simulate various hypothetical management scenarios in a context of forest insect infestations. The social and ecological components of this model are developed in two different environments. In order to establish the connection between those components, this model is equipped with a ‘FlipFlop’ - a structure of storage directories and communication protocols which allows each of the models to process its inputs, send an output message to the other, and/or wait for an input message from the other, when necessary. To see the publications associated with the social and ecological components of this coupled model please see the References section.

Our model shows how disinformation spreads on a random network of individuals. The network is weighted and directed. We are looking at how different factors affect how fast, or how many people get “infected” with the misinformation. One of the main factors that we were curious about was perceived trustworthiness. This is because we want to see if people of power, or a high degree of perceived trustworthiness, were able to push misinformation to more people and convert more people to believe the information.

This model implements two types of network diffusion from an initial group of activated nodes. In complex contagion, a node is activated if the proportion of neighbour nodes that are already activated exceeds a given threshold. This is intended to represented the spread of health behaviours. In simple contagion, an activated node has a given probability of activating its inactive neighbours and re-tests each time step until all of the neighbours are activated. This is intended to represent information spread.

A range of networks are included with the model from secondary school friendship networks. The proportion of nodes initially activated and the method of selecting those nodes are controlled by the user.

This is an extension of the basic Suceptible, Infected, Recovered (SIR) model. This model explores the spread of disease in two spaces, one a treatment, and one a control. Through the modeling options, one can explore how changing assumptions about the number of susceptible people, starting number of infected people, the disease’s infection probability, and average duration impacts the outcome. In addition, this version allows users to explore how public health interventions like social distancing, masking, and isolation can affect the number of people infected. The model shows that the interactions of agents, and the interventions can drastically affect the results of the model.

We used the model in our course about COVID-19: https://www.csats.psu.edu/science-of-covid19

This is a basic Susceptible, Infected, Recovered (SIR) model. This model explores the spread of disease in a space. In particular, it explores how changing assumptions about the number of susceptible people, starting number of infected people, as well as the disease’s infection probability, and average duration of infection. The model shows that the interactions of agents can drastically affect the results of the model.

We used it in our course on COVID-19: https://www.csats.psu.edu/science-of-covid19

Multi-layer network agent-based model of the progression of the COVID19 infection, digital contact tracing

A curious aspect of the Covid-19 pandemic is the clustering of outbreaks. Evidence suggests that 80\% of people who contract the virus are infected by only 19% of infected individuals, and that the majority of infected individuals faile to infect another person. Thus, the dispersion of a contagion, $k$, may be of more use in understanding the spread of Covid-19 than the reproduction number, R0.

The Virus Transmission with Super-spreaders model, written in NetLogo, is an adaptation of the canonical Virus Transmission on a Network model and allows the exploration of various mitigation protocols such as testing and quarantines with both homogenous transmission and heterogenous transmission.

The model consists of a population of individuals arranged in a network, where both population and network degree are tunable. At the start of the simulation, a subset of the population is initially infected. As the model runs, infected individuals will infect neighboring susceptible individuals according to either homogenous or heterogenous transmission, where heterogenous transmission models super-spreaders. In this case, k is described as the percentage of super-spreaders in the population and the differing transmission rates for super-spreaders and non super-spreaders. Infected individuals either recover, at which point they become resistant to infection, or die. Testing regimes cause discovered infected individuals to quarantine for a period of time.