CoMSES Net maintains cyberinfrastructure to foster FAIR data principles for access to and (re)use of computational models. Model authors can publish their model code in the Computational Model Library with documentation, metadata, and data dependencies and support these FAIR data principles as well as best practices for software citation. Model authors can also request that their model code be peer reviewed to receive a DOI. All users of models published in the library must cite model authors when they use and benefit from their code.
CoMSES Net also maintains a curated database of over 7500 publications of agent-based and individual based models with additional metadata on availability of code and bibliometric information on the landscape of ABM/IBM publications that we welcome you to explore.
This paper tries to shed some light on the mutual influence of citizen behaviour and the spread of a virus in an epidemic. While the spread of a virus from infectious to susceptible persons and the outbreak of an infection leading to more or less severe illness and, finally, to recovery and immunity or death has been modelled with different kinds of models in the past, the influence of certain behaviours to keep the epidemic low and to follow recommendations of others to apply these behaviours has rarely been modelled. The model introduced here uses a theory of the effect of norm invocations among persons to find out the effect of spreading norms interacts with the progress of an epidemic. Results show that norm invocations matter. The model replicates the histories of the COVID-19 epidemic in various region, including “second waves”, and shows that the calculation of the reproduction numbers from current reported infections usually overestimates the “real” but in practice unobservable reproduction number.
A curious aspect of the Covid-19 pandemic is the clustering of outbreaks. Evidence suggests that 80\% of people who contract the virus are infected by only 19% of infected individuals, and that the majority of infected individuals faile to infect another person. Thus, the dispersion of a contagion, $k$, may be of more use in understanding the spread of Covid-19 than the reproduction number, R0.
The Virus Transmission with Super-spreaders model, written in NetLogo, is an adaptation of the canonical Virus Transmission on a Network model and allows the exploration of various mitigation protocols such as testing and quarantines with both homogenous transmission and heterogenous transmission.
The model consists of a population of individuals arranged in a network, where both population and network degree are tunable. At the start of the simulation, a subset of the population is initially infected. As the model runs, infected individuals will infect neighboring susceptible individuals according to either homogenous or heterogenous transmission, where heterogenous transmission models super-spreaders. In this case, k is described as the percentage of super-spreaders in the population and the differing transmission rates for super-spreaders and non super-spreaders. Infected individuals either recover, at which point they become resistant to infection, or die. Testing regimes cause discovered infected individuals to quarantine for a period of time.
A minimal genetic algorithm was preliminarily developed to search for the solution of an elementary arithmetic problem. It has been modified to explore the effect of a mutator gene and the consequent entrance into a hypermutation state. The phenomenon is particularly important in some types of tumorigenesis and in a more general way, in cells and tissues submitted to chronic sublethal environmental or genomic stress.
Since a long time, some scholars suppose that organisms speed up their own evolution by varying mutation rate, but evolutionary biologists are not convinced that evolution can select a mechanism promoting more (often harmful) mutations looking forward an environmental challenge.
The model aims to shed light on these controversial points of view and it provides also the features required to check the role of sex and genetic recombination in the mutator genes diffusion.
The three-day participatory workshop organized by the TISSS Lab had 20 participants who were academics in different career stages ranging from university student to professor. For each of the five games, the participants had to move between tables according to some pre-specified rules. After the workshop both the participant’s perception of the games’ complexities and the participants’ satisfaction with the games were recorded.
In order to obtain additional objective measures for the games’ complexities, these games were also simulated using this simulation model here. Therefore, the simulation model is an as-accurate-as-possible reproduction of the workshop games: it has 20 participants moving between 5 different tables. The rules that specify who moves when vary from game to game. Just to get an idea, Game 3 has the rule: “move if you’re sitting next to someone who is waring white or no socks”.
An exact description of the workshop games and the associated simulation models can be found in the paper “The relation between perceived complexity and happiness with decision situations: searching for objective measures in social simulation games”.
RAGE models a stylized common property grazing system. Agents follow a certain behavioral type. The model allows analyzing how household behavior with respect to a social norm on pasture resting affects long-term social-ecological system dynamics.
Evolution of Sex is a NetLogo model that illustrates the advantages and disadvantages of sexual and asexual reproductive strategies. It seeks to demonstrate the answer to the question “Why do we have sex?”
This is a variation of the Sugarspace model of Axtell and Epstein (1996) with spice and trade of sugar and spice. The model is not an exact replication since we have a somewhat simpler landscape of sugar and spice resources included, as well as a simple reproduction rule where agents with a certain accumulated wealth derive an offspring (if a nearby empty patch is available).
The model is discussed in Introduction to Agent-Based Modeling by Marco Janssen. For more information see https://intro2abm.com/
This agent-based model was built as part of a replication effort of Jeness et al.’s work (linked below). The model simulates an MSM sexual activity network for the purpose of modeling the effects of respectively PrEP and ART on HIV prevention. The purpose of the model is to explore the differences between differerent interpretations of the NIH Indication Guidelines for PrEP.
This is a modification of a model published previous by Barton and Riel-Salvatore (2012). In this model, we simulate six regional populations within Last Glacial Maximum western Europe. Agents interact through reproduction and genetic markers attached to each of six regions mix through subsequent generations as a way to track population dynamics, mobility, and gene flow. In addition, the landscape is heterogeneous and affects agent mobility and, under certain scenarios, their odds of survival.
We compare three model estimates for the time and treatment requirements to eliminate HCV among HIV-positive MSM in Victoria, Australia: a compartmental model; an ABM parametrized by surveillance data; and an ABM with a more heterogeneous population.