CoMSES Net maintains cyberinfrastructure to foster FAIR data principles for access to and (re)use of computational models. Model authors can publish their model code in the Computational Model Library with documentation, metadata, and data dependencies and support these FAIR data principles as well as best practices for software citation. Model authors can also request that their model code be peer reviewed to receive a DOI. All users of models published in the library must cite model authors when they use and benefit from their code.
CoMSES Net also maintains a curated database of over 7500 publications of agent-based and individual based models with additional metadata on availability of code and bibliometric information on the landscape of ABM/IBM publications that we welcome you to explore.
This paper tries to shed some light on the mutual influence of citizen behaviour and the spread of a virus in an epidemic. While the spread of a virus from infectious to susceptible persons and the outbreak of an infection leading to more or less severe illness and, finally, to recovery and immunity or death has been modelled with different kinds of models in the past, the influence of certain behaviours to keep the epidemic low and to follow recommendations of others to apply these behaviours has rarely been modelled. The model introduced here uses a theory of the effect of norm invocations among persons to find out the effect of spreading norms interacts with the progress of an epidemic. Results show that norm invocations matter. The model replicates the histories of the COVID-19 epidemic in various region, including “second waves”, and shows that the calculation of the reproduction numbers from current reported infections usually overestimates the “real” but in practice unobservable reproduction number.
The Multilevel Group Selection I (MGS I) model simulates a population of contributing and non-contributing agents, competing on a social landscape for higher-value spots in an effort to withstand some selection pressure. It may be useful to both scientists and students in hypothesis testing, theory development, or more generally in understanding multilevel group selection.
EMMIT is an end-user developed agent-based simulation of malaria transmission. The simulation’s development is a case study demonstrating an approach for non-technical investigators to easily develop useful simulations of complex public health problems. We focused on malaria transmission, a major global public health problem, and insecticide resistance (IR), a major problem affecting malaria control. Insecticides are used to reduce transmission of malaria caused by the Plasmodium parasite that is spread by the Anopheles mosquito. However, the emergence and spread of IR in a mosquito population can diminish the insecticide’s effectiveness. IR results from mutations that produce behavioral changes or biochemical changes (such as detoxification enhancement, target site alterations) in the mosquito population that provide resistance to the insecticide. Evolutionary selection for the IR traits reduces the effectiveness of an insecticide favoring the resistant mosquito population. It has been suggested that biopesticides, and specifically those that are Late Life Acting (LLA), could address this problem. LLA insecticides exploit Plasmodium’s approximate 10-day extrinsic incubation period in the mosquito vector, a delay that limits malaria transmission to older infected mosquitoes. Since the proposed LLA insecticide delays mosquito death until after the exposed mosquito has a chance to produce several broods of offspring, reducing the selective pressure for resistance, it delays IR development and gives the insecticide longer effectivity. Such insecticides are designed to slow the evolution of IR thus maintaining their effectiveness for malaria control. For the IR problem, EMMIT shows that an LLA insecticide could work as intended, but its operational characteristics are critical, primarily the mean-time-to-death after exposure and the associated standard deviation. We also demonstrate the simulation’s extensibility to other malaria control measures, including larval source control and policies to mitigate the spread of IR. The simulation was developed using NetLogo as a case study of a simple but useful approach to public health research.
Ecosystems are among the most complex structures studied. They comprise elements that seem both stable and contingent. The stability of these systems depends on interactions among their evolutionary history, including the accidents of organisms moving through the landscape and microhabitats of the earth, and the biotic and abiotic conditions in which they occur. When ecosystems are stable, how is that achieved? Here we look at ecosystem stability through a computer simulation model that suggests that it may depend on what constrains the system and how those constraints are structured. Specifically, if the constraints found in an ecological community form a closed loop, that allows particular kinds of feedback may give structure to the ecosystem processes for a period of time. In this simulation model, we look at how evolutionary forces act in such a way these closed constraint loops may form. This may explain some kinds of ecosystem stability. This work will also be valuable to ecological theorists in understanding general ideas of stability in such systems.
This model is a replication model which is constructed based on the existing model used by the following article:
Brown, D.G. and Robinson, D.T., 2006. Effects of heterogeneity in residential preferences on an agent-based model of urban sprawl. Ecology and society, 11(1).
The original model is called SLUCE’s Original Model for Experimentation (SOME). In Brown and Robinson (2006)’s article, the SOME model was used to explore the impacts of heterogeneity in residential location selections on the research of urban sprawl. The original model was constructed using Objective-C language based on SWARM platform. This replication model is built by NetLogo language on NetLogo platform. We successfully replicate that model and demonstrated the reliability and replicability of it.
PopComp by Andre Costopoulos 2020
Licence: DWYWWI (Do whatever you want with it)
I use Netlogo to build a simple environmental change and population expansion and diffusion model. Patches have a carrying capacity and can host two kinds of populations (APop and BPop). Each time step, the carrying capacity of each patch has a given probability of increasing or decreasing up to a maximum proportion.
This model simulates different seeding strategies for information diffusion in a social network adjusted to a case study area in rural Zambia. It systematically evaluates different criteria for seed selection (centrality measures and hierarchy), number of seeds, and interaction effects between seed selection criteria and set size.
B3GET simulates populations of virtual organisms evolving over generations, whose evolutionary outcomes reflect the selection pressures of their environment. The model simulates several factors considered important in biology, including life history trade-offs, investment in fighting ability and aggression, sperm competition, infanticide, and competition over access to food and mates. Downloaded materials include a starting genotype and population files. Edit the these files and see what changes occur in the behavior of virtual populations!
The model simulates agents in a spatial environment competing for a common resource that grows on patches. The resource is converted to energy, which is needed for performing actions and for surviving.
This model simulates the mechanisms of evolution, or how allele frequencies change in a population over time.