CoMSES Net maintains cyberinfrastructure to foster FAIR data principles for access to and (re)use of computational models. Model authors can publish their model code in the Computational Model Library with documentation, metadata, and data dependencies and support these FAIR data principles as well as best practices for software citation. Model authors can also request that their model code be peer reviewed to receive a DOI. All users of models published in the library must cite model authors when they use and benefit from their code.
CoMSES Net also maintains a curated database of over 7500 publications of agent-based and individual based models with additional metadata on availability of code and bibliometric information on the landscape of ABM/IBM publications that we welcome you to explore.
Ecosystems are among the most complex structures studied. They comprise elements that seem both stable and contingent. The stability of these systems depends on interactions among their evolutionary history, including the accidents of organisms moving through the landscape and microhabitats of the earth, and the biotic and abiotic conditions in which they occur. When ecosystems are stable, how is that achieved? Here we look at ecosystem stability through a computer simulation model that suggests that it may depend on what constrains the system and how those constraints are structured. Specifically, if the constraints found in an ecological community form a closed loop, that allows particular kinds of feedback may give structure to the ecosystem processes for a period of time. In this simulation model, we look at how evolutionary forces act in such a way these closed constraint loops may form. This may explain some kinds of ecosystem stability. This work will also be valuable to ecological theorists in understanding general ideas of stability in such systems.
B3GET simulates populations of virtual organisms evolving over generations, whose evolutionary outcomes reflect the selection pressures of their environment. The model simulates several factors considered important in biology, including life history trade-offs, investment in fighting ability and aggression, sperm competition, infanticide, and competition over access to food and mates. Downloaded materials include a starting genotype and population files. Edit the these files and see what changes occur in the behavior of virtual populations!
Implementation of Milbrath’s (1965) model of political participation. Individual participation is determined by stimuli from the political environment, interpersonal interaction, as well as individual characteristics.
The model is a combination of a spatially explicit, stochastic, agent-based model for wild boars (Sus scrofa L.) and an epidemiological model for the Classical Swine Fever (CSF) virus infecting the wild boars.
The original model (Kramer-Schadt et al. 2009) was used to assess intrinsic (system immanent host-pathogen interaction and host life-history) and extrinsic (spatial extent and density) factors contributing to the long-term persistence of the disease and has further been used to assess the effects of intrinsic dynamics (Lange et al. 2012a) and indirect transmission (Lange et al. 2016) on the disease course. In an applied context, the model was used to test the efficiency of spatiotemporal vaccination regimes (Lange et al. 2012b) as well as the risk of disease spread in the country of Denmark (Alban et al. 2005).
References: See ODD model description.
A model that allows for representing key theories of Roman amphora reuse, to explore the differences in the distribution of amphorae, re-used amphorae and their contents.
This model generates simulated distributions of prime-use amphorae, primeuse contents (e.g. olive oil) and reused amphorae. These simulated distributions will differ between experiments depending on the experiment’s variable settings representing the tested theory: variations in the probability of reuse, the supply volume, the probability of reuse at ports. What we are interested in teasing out is what the effect is of each theory on the simulated amphora distributions.
The results presented in the related publication (Brughmans and Pecci in press) for all experiments were obtained after running the simulation for 1000 time steps, at which point the simulated distribution patterns have stabilized.
The model is an agent-based artificial stock market where investors connect in a dynamic network. The network is dynamic in the sense that the investors, at specified intervals, decide whether to keep their current adviser (those investors they receive trading advise from). The investors also gain information from a private source and share public information about the risky asset. Investors have different tendencies to follow the different information sources, consider differing amounts of history, and have different thresholds for investing.
This model is an extended version of the original MERCURY model (https://www.comses.net/codebases/4347/releases/1.1.0/ ) . It allows for experiments to be performed in which empirically informed population sizes of sites are included, that allow for the scaling of the number of tableware traders with the population of settlements, and for hypothesised production centres of four tablewares to be used in experiments.
Experiments performed with this population extension and substantive interpretations derived from them are published in:
Hanson, J.W. & T. Brughmans. In press. Settlement scale and economic networks in the Roman Empire, in T. Brughmans & A.I. Wilson (ed.) Simulating Roman Economies. Theories, Methods and Computational Models. Oxford: Oxford University Press.
This paper builds on a basic ABM for a revolution and adds a combination of behaviors to its agents such as military benefits, citizen’s grievances, geographic vision, empathy, personality type and media impact.
A global model of the 1918-19 Influenza Pandemic. It can be run to match history or explore counterfactual questions about the influence of World War I on the dynamics of the epidemic. Explores two theories of the location of the initial infection.